New evidence of the impact of mitochondria on kidney health and disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Nephrology
Link
https://www.nature.com/articles/s41581-023-00803-z.pdf
Reference6 articles.
1. Clark, A. J., Saade, M. C. & Parikh, S. M. The significance of NAD+ biosynthesis alterations in acute kidney injury. Semin. Nephrol. 42, 151287 (2022).
2. Doke, T. et al. NAD+ precursor supplementation prevents mtRNA/RIG-I-dependent inflammation during kidney injury. Nat. Metab. 5, 414–430 (2023).
3. Morel, J.-D. et al. Mitochondrial and NAD+ metabolism predict recovery from acute kidney injury in a diverse mouse population. JCI Insight 8, e164626 (2023).
4. Verissimo, T. et al. PCK1 is a key regulator of metabolic and mitochondrial functions in renal tubular cells. Am. J. Physiol. Renal Physiol. 324, F532–F543 (2023).
5. Hasegawa, K., Sakamaki, Y., Tamaki, M. & Wakino, S. PCK1 protects against mitoribosomal defects in diabetic nephropathy in mouse models. J. Am. Soc. Nephrol. 34, 1343–1365 (2023).
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