Sodium selenate regulates the brain ionome in a transgenic mouse model of Alzheimer’s disease-Reference-Cited by-同舟云学术

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Sodium selenate regulates the brain ionome in a transgenic mouse model of Alzheimer’s disease

Published:2016-12 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Zheng Lin,Zhu Hua-Zhang,Wang Bing-Tao,Zhao Qiong-Hui,Du Xiu-Bo,Zheng Yi,Jiang Liang,Ni Jia-Zuan,Zhang Yan,Liu Qiong

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep39290.pdf

Reference83 articles.

1. Lewczuk, P., Mroczko, B., Fagan, A. & Kornhuber, J. Biomarkers of Alzheimer’s disease and mild cognitive impairment: a current perspective. Advances in medical sciences 60, 76–82, doi: 10.1016/j.advms.2014.11.002 (2015).

2. Spires-Jones, T. L. & Hyman, B. T. The intersection of amyloid beta and tau at synapses in Alzheimer’s disease. Neuron 82, 756–771, doi: 10.1016/j.neuron.2014.05.004 (2014).

3. Hubin, E., van Nuland, N. A., Broersen, K. & Pauwels, K. Transient dynamics of Abeta contribute to toxicity in Alzheimer’s disease. Cellular and molecular life sciences: CMLS 71, 3507–3521, doi: 10.1007/s00018-014-1634-z (2014).

4. Sato, N. & Morishita, R. Brain alterations and clinical symptoms of dementia in diabetes: abeta/tau-dependent and independent mechanisms. Frontiers in endocrinology 5, 143, doi: 10.3389/fendo.2014.00143 (2014).

5. Liao, D., Miller, E. C. & Teravskis, P. J. Tau acts as a mediator for Alzheimer’s disease-related synaptic deficits. The European journal of neuroscience 39, 1202–1213, doi: 10.1111/ejn.12504 (2014).

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