PD-L1 and PD-L2 modulate airway inflammation and iNKT-cell-dependent airway hyperreactivity in opposing directions-Reference-Cited by-同舟云学术

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PD-L1 and PD-L2 modulate airway inflammation and iNKT-cell-dependent airway hyperreactivity in opposing directions

Published:2009-09-09 Issue:1 Volume:3 Page:81-91
ISSN:1933-0219
Container-title:Mucosal Immunology
language:en
Short-container-title:Mucosal Immunol

Author:

Akbari O,Stock P,Singh A K,Lombardi V,Lee W-L,Freeman G J,Sharpe A H,Umetsu D T,DeKruyff R H

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/mi2009112.pdf

Reference49 articles.

1. Deppong, C. et al. Cutting edge: B and T lymphocyte attenuator and programmed death receptor-1 inhibitory receptors are required for termination of acute allergic airway inflammation. J. Immunol. 176, 3909–3913 (2006).

2. Akbari, O. et al. Essential role of NKT cells producing IL-4 and IL-13 in the development of allergen-induced airway hyperreactivity. Nat. Med. 9, 582–588 (2003).

3. Lisbonne, M. et al. Cutting edge: invariant V alpha 14 NKT cells are required for allergen-induced airway inflammation and hyperreactivity in an experimental asthma model. J. Immunol. 171, 1637–1641 (2003).

4. Jin, N. et al. Airway hyperresponsiveness through synergy of {gamma}{delta} T cells and NKT cells. J. Immunol. 179, 2961–2968 (2007).

5. Meyer, E.H. et al. Glycolipid activation of invariant T cell receptor + NK T cells is sufficient to induce airway hyperreactivity independent of conventional CD4+ T cells. Proc. Natl. Acad. Sci. USA 103, 2782–2787 (2006).

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