Mechanism of nonhomologous end-joining in mycobacteria: a low-fidelity repair system driven by Ku, ligase D and ligase C-Reference-Cited by-同舟云学术

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Mechanism of nonhomologous end-joining in mycobacteria: a low-fidelity repair system driven by Ku, ligase D and ligase C

Published:2005-03-20 Issue:4 Volume:12 Page:304-312
ISSN:1545-9993
Container-title:Nature Structural & Molecular Biology
language:en
Short-container-title:Nat Struct Mol Biol

Author:

Gong Chunling,Bongiorno Paola,Martins Alexandra,Stephanou Nicolas C,Zhu Hui,Shuman Stewart,Glickman Michael S

Publisher

Springer Science and Business Media LLC

Subject

Molecular Biology,Structural Biology

Link

http://www.nature.com/articles/nsmb915.pdf

Reference39 articles.

1. Buchmeier, N.A. et al. DNA repair is more important than catalase for Salmonella virulence in mice. J. Clin. Invest. 95, 1047–1053 (1995).

2. Buchmeier, N.A., Lipps, C.J., So, M.Y. & Heffron, F. Recombination-deficient mutants of Salmonella typhimurium are avirulent and sensitive to the oxidative burst of macrophages. Mol. Microbiol. 7, 933–936 (1993).

3. O'Rourke, E.J. et al. Pathogen DNA as target for host-generated oxidative stress: role for repair of bacterial DNA damage in Helicobacter pylori colonization. Proc. Natl. Acad. Sci. USA 100, 2789–2794 (2003).

4. Suvarnapunya, A.E., Lagasse, H.A. & Stein, M.A. The role of DNA base excision repair in the pathogenesis of Salmonella enterica serovar Typhimurium. Mol. Microbiol. 48, 549–559 (2003).

5. Sander, P. et al. Mycobacterium bovis BCG recA deletion mutant shows increased susceptibility to DNA-damaging agents but wild-type survival in a mouse infection model. Infect. Immun. 69, 3562–3568 (2001).

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