Bmf is a possible mediator in histone deacetylase inhibitors FK228 and CBHA-induced apoptosis

Author:

Zhang Y,Adachi M,Kawamura R,Imai K

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Reference38 articles.

1. Kouzarides T (2000) Acetylation: a regulatory modification to rival phosphorylation? EMBO J. 19: 1176–1179

2. Yoshida M and Horinouchi S (1999) Trichostatin and leptomycin. Inhibition of histone deacetylation and signal-dependent nuclear export. Ann. NY. Acad. Sci. 886: 23–36

3. Marks PA, Richon VM and Rifkind RA (2000) Histone deacetylase inhibitors: inducers of differentiation or apoptosis of transformed cells. J. Natl. Cancer Inst. 92: 1210–1216

4. Nervi C, Borello U, Fazi F, Buffa V, Pelicci PG and Cossu G (2001) Inhibition of histone deacetylase activity by trichostatin A modulates gene expression during mouse embryogenesis without apparent toxicity. Cancer Res. 61: 1247–1249

5. Goldsmith ME, Kitazono M, Fok P, Aikou T, Bates S and Fojo T (2003) The histone deacetylase inhibitor FK228 preferentially enhances adenovirus transgene expression in malignant cells. Clin. Cancer Res. 9: 5394–5401

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