The Cdh5-CreERT2 transgene causes conditional Shb gene deletion in hematopoietic cells with consequences for immune cell responses to tumors-Reference-Cited by-同舟云学术

The Cdh5-CreERT2 transgene causes conditional Shb gene deletion in hematopoietic cells with consequences for immune cell responses to tumors

Published:2019-05-17 Issue:1 Volume:9 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

He Qi,Li Xiujuan^ORCID,Singh Kailash^ORCID,Luo Zhengkang,Meija-Cordova Mariela,Jamalpour Maria,Lindahl Björn^ORCID,Kriz Vitezslav,Vuolteenaho Reetta,Ulvmar Maria^ORCID,Welsh Michael^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-019-44039-z.pdf

Reference33 articles.

1. Welsh, M., Jamalpour, M., Zang, G. & Akerblom, B. The role of the Src Homology-2 domain containing protein B (SHB) in beta cells. J Mol Endocrinol 56, R21–31, https://doi.org/10.1530/JME-15-0228 (2016).

2. Funa, N. S. et al. Dysfunctional microvasculature as a consequence of shb gene inactivation causes impaired tumor growth. Cancer research 69, 2141–2148, https://doi.org/10.1158/0008-5472.CAN-08-3797 (2009).

3. Akerblom, B. et al. Heterogeneity among RIP-Tag2 insulinomas allows vascular endothelial growth factor-A independent tumor expansion as revealed by studies in Shb mutant mice: implications for tumor angiogenesis. Molecular oncology 6, 333–346, https://doi.org/10.1016/j.molonc.2012.01.006 (2012).

4. Zang, G. et al. Vascular dysfunction and increased metastasis of B16F10 melanomas in Shb deficient mice as compared with their wild type counterparts. BMC Cancer 15, 234, https://doi.org/10.1186/s12885-015-1269-y (2015).

5. Li, X. et al. Pro-tumoral immune cell alterations in wild type and Shb-deficient mice in response to 4T1 breast carcinomas. Oncotarget 9, 18720–18733, https://doi.org/10.18632/oncotarget.24643 (2018).

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