Non-alcoholic fatty liver disease, vascular inflammation and insulin resistance are exacerbated by TRAIL deletion in mice-Reference-Cited by-同舟云学术

Non-alcoholic fatty liver disease, vascular inflammation and insulin resistance are exacerbated by TRAIL deletion in mice

Published:2017-05-15 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Cartland Siân P.^ORCID,Harith Hanis H.,Genner Scott W.,Dang Lei,Cogger Victoria C.,Vellozzi Melissa,Di Bartolo Belinda A.,Thomas Shane R.,Adams Leon A.,Kavurma Mary M.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-01721-4.pdf

Reference52 articles.

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2. Wong, R. J. et al. Nonalcoholic steatohepatitis is the second leading etiology of liver disease among adults awaiting liver transplantation in the United States. Gastroenterology 148, 547–555, doi: 10.1053/j.gastro.2014.11.039 (2015).

3. Smith, B. W. & Adams, L. A. Non-alcoholic fatty liver disease. Crit Rev Clin Lab Sci 48, 97–113, doi: 10.3109/10408363.2011.596521 (2011).

4. Azahri, N. S. & Kavurma, M. M. Transcriptional regulation of tumour necrosis factor-related apoptosis-inducing ligand. Cellular and molecular life sciences: CMLS 70, 3617–3629, doi: 10.1007/s00018-013-1264-x (2013).

5. Kavurma, M. M. & Bennett, M. R. Expression, regulation and function of trail in atherosclerosis. Biochem Pharmacol 75, 1441–1450, doi: 10.1016/j.bcp.2007.10.020 (2008).

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