Selective targeting of Scn8a prevents seizure development in a mouse model of mesial temporal lobe epilepsy-Reference-Cited by-同舟云学术

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Selective targeting of Scn8a prevents seizure development in a mouse model of mesial temporal lobe epilepsy

Published:2018-01-09 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Wong Jennifer C.,Makinson Christopher D.,Lamar Tyra,Cheng Qi,Wingard Jeffrey C.,Terwilliger Ernest F.,Escayg Andrew

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-17786-0.pdf

Reference58 articles.

1. Brodie, M. J. et al. Commission on European Affairs: appropriate standards of epilepsy care across Europe.ILEA. Epilepsia 38, 1245–1250 (1997).

2. Kwan, P. & Brodie, M. J. Early identification of refractory epilepsy. The New England journal of medicine 342, 314–319, https://doi.org/10.1056/NEJM200002033420503 (2000).

3. Raman, I. M., Sprunger, L. K., Meisler, M. H. & Bean, B. P. Altered subthreshold sodium currents and disrupted firing patterns in Purkinje neurons of Scn8a mutant mice. Neuron 19, 881–891 (1997).

4. Martin, M. S. et al. The voltage-gated sodium channel Scn8a is a genetic modifier of severe myoclonic epilepsy of infancy. Human molecular genetics 16, 2892–2899, https://doi.org/10.1093/hmg/ddm248 (2007).

5. Blumenfeld, H. et al. Role of hippocampal sodium channel Nav1.6 in kindling epileptogenesis. Epilepsia 50, 44–55, https://doi.org/10.1111/j.1528-1167.2008.01710.x (2009).

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