Experimental Metabolic Syndrome Model Associated with Mechanical and Structural Degenerative Changes of the Aortic Valve
Author:
Funder
Foundation for the National Institutes of Health
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-36388-y.pdf
Reference51 articles.
1. Caira, F. C. et al. Human degenerative valve disease is associated with up-regulation of low-density lipoprotein receptor-related protein 5 receptor-mediated bone formation. Journal of the American College of Cardiology 47, 1707–1712, https://doi.org/10.1016/j.jacc.2006.02.040 (2006).
2. Rajamannan, N. M. et al. Human aortic valve calcification is associated with an osteoblast phenotype. Circulation 107, 2181–2184, https://doi.org/10.1161/01.cir.0000070591.21548.69 (2003).
3. Mahmut, A. et al. Elevated expression of lipoprotein-associated phospholipase A2 in calcific aortic valve disease: implications for valve mineralization. Journal of the American College of Cardiology 63, 460–469, https://doi.org/10.1016/j.jacc.2013.05.105 (2014).
4. Mahmut, A. et al. Lp-PLA2 is associated with structural valve degeneration of bioprostheses. European journal of clinical investigation 44, 136–145, https://doi.org/10.1111/eci.12199 (2014).
5. Lindman, B. R. et al. Calcific aortic stenosis. Nature reviews. Disease primers 2, 16006, https://doi.org/10.1038/nrdp.2016.6 (2016).
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