Inactive Tlk associating with Tak1 increases p38 MAPK activity to prolong the G2 phase
Author:
Funder
Ministry of Science and Technology, Taiwan
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-36137-1.pdf
Reference60 articles.
1. Vijg, J. & Suh, Y. Genome instability and aging. Annu Rev Physiol. 75, 645–668, https://doi.org/10.1146/annurev-physiol-030212-183715 (2013).
2. Bulavin, D. V. et al. Initiation of a G2/M checkpoint after ultraviolet radiation requires p38 kinase. Nature 411, 102–107, https://doi.org/10.1038/35075107 (2001).
3. Bruinsma, W., van den Berg, J., Aprelia, M. & Medema, R. H. Tousled-like kinase 2 regulates recovery from a DNA damage-induced G2 arrest. EMBO Rep. 17, 659–670, https://doi.org/10.15252/embr.201540767 (2016).
4. Kelly, R. & Davey, S. K. Tousled-like kinase-dependent phosphorylation of Rad9 plays a role in cell cycle progression and G2/M checkpoint exit. PLoS One 8, e85859, https://doi.org/10.1371/journal.pone.0085859 (2013).
5. Sunavala-Dossabhoy, G. & De Benedetti, A. Tousled homolog, TLK1, binds and phosphorylates Rad9; TLK1 acts as a molecular chaperone in DNA repair. DNA Repair 8, 87–102, https://doi.org/10.1016/j.dnarep.2008.09.005 (2009).
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