Importance of the novel organic cation transporter 1 for tyrosine kinase inhibition by saracatinib in rheumatoid arthritis synovial fibroblasts
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-01438-4.pdf
Reference43 articles.
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2. Huber, L. C. et al. Synovial fibroblasts: key players in rheumatoid arthritis. Rheumatology (Oxford) 45, 669–675, doi: 10.1093/rheumatology/kel065 (2006).
3. Korb-Pap, A., Bertrand, J., Sherwood, J. & Pap, T. Stable activation of fibroblasts in rheumatic arthritis-causes and consequences. Rheumatology (Oxford) 55, ii64–ii67, doi: 10.1093/rheumatology/kew347 (2016).
4. Mocsai, A., Kovacs, L. & Gergely, P. What is the future of targeted therapy in rheumatology: biologics or small molecules? BMC Med 12, 43, doi: 10.1186/1741-7015-12-43 (2014).
5. Meier, F. M. & McInnes, I. B. Small-molecule therapeutics in rheumatoid arthritis: Scientific rationale, efficacy and safety. Best Pract Res Clin Rheumatol 28, 605–624, doi: 10.1016/j.berh.2014.10.017 (2014).
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