Human erythrocytes release ATP by a novel pathway involving VDAC oligomerization independent of pannexin-1
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-29885-7.pdf
Reference64 articles.
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2. Olearczyk, J. J., Ellsworth, M. L., Stephenson, A. H., Lonigro, A. J. & Sprague, R. S. Nitric oxide inhibits ATP release from erythrocytes. The Journal of pharmacology and experimental therapeutics 309, 1079–1084, https://doi.org/10.1124/jpet.103.064709 (2004).
3. Sprague, R. S., Ellsworth, M. L., Stephenson, A. H. & Lonigro, A. J. Participation of cAMP in a signal-transduction pathway relating erythrocyte deformation to ATP release. American journal of physiology. Cell physiology 281, C1158–1164 (2001).
4. Sikora, J., Orlov, S. N., Furuya, K. & Grygorczyk, R. Hemolysis is a primary ATP-release mechanism in human erythrocytes. Blood 124, 2150–2157, https://doi.org/10.1182/blood-2014-05-572024 (2014).
5. Lazarowski, E. R. Vesicular and conductive mechanisms of nucleotide release. Purinergic signalling 8, 359–373, https://doi.org/10.1007/s11302-012-9304-9 (2012).
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