Myocardial hypothermia increases autophagic flux, mitochondrial mass and myocardial function after ischemia-reperfusion injury-Reference-Cited by-同舟云学术

Myocardial hypothermia increases autophagic flux, mitochondrial mass and myocardial function after ischemia-reperfusion injury

Published:2019-07-10 Issue:1 Volume:9 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Marek-Iannucci Stefanie,Thomas Amandine,Hou Jean,Crupi Annunziata,Sin Jon,Taylor David J.,Czer Lawrence S.^ORCID,Esmailian Fardad,Mentzer Robert M.,Andres Allen M.,Gottlieb Roberta A.^ORCID

Funder

U.S. Department of Health & Human Services | National Institutes of Health

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-019-46452-w.pdf

Reference31 articles.

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3. Shi, J., Dai, W. & Kloner, R. A. Therapeutic Hypothermia Reduces the Inflammatory Response Following Ischemia/Reperfusion Injury in Rat Hearts. Ther Hypothermia Temp Manag. 7, 162–170 (2017).

4. Dash, R. et al. Dose-Dependent Cardioprotection of Moderate (32 °C) Versus Mild (35 °C) Therapeutic Hypothermia in Porcine Acute Myocardial Infarction. JACC Cardiovasc Interv. 11, 195–205 (2018).

5. Hale, S. L. & Kloner, R. A. Mild hypothermia as a cardioprotective approach for acute myocardial infarction: laboratory to clinical application. J Cardiovasc Pharmacol Ther. 16, 131–9 (2011).

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