Myocardial hypothermia increases autophagic flux, mitochondrial mass and myocardial function after ischemia-reperfusion injury
Author:
Funder
U.S. Department of Health & Human Services | National Institutes of Health
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-019-46452-w.pdf
Reference31 articles.
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3. Shi, J., Dai, W. & Kloner, R. A. Therapeutic Hypothermia Reduces the Inflammatory Response Following Ischemia/Reperfusion Injury in Rat Hearts. Ther Hypothermia Temp Manag. 7, 162–170 (2017).
4. Dash, R. et al. Dose-Dependent Cardioprotection of Moderate (32 °C) Versus Mild (35 °C) Therapeutic Hypothermia in Porcine Acute Myocardial Infarction. JACC Cardiovasc Interv. 11, 195–205 (2018).
5. Hale, S. L. & Kloner, R. A. Mild hypothermia as a cardioprotective approach for acute myocardial infarction: laboratory to clinical application. J Cardiovasc Pharmacol Ther. 16, 131–9 (2011).
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