Granzyme B is elevated in autoimmune blistering diseases and cleaves key anchoring proteins of the dermal-epidermal junction-Reference-Cited by-同舟云学术

Granzyme B is elevated in autoimmune blistering diseases and cleaves key anchoring proteins of the dermal-epidermal junction

Published:2018-06-26 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Russo Valerio,Klein Theo^ORCID,Lim Darielle J.,Solis Nestor,Machado Yoan,Hiroyasu Sho,Nabai Layla,Shen Yue,Zeglinski Matthew R.,Zhao Hongyan,Oram Cameron P.,Lennox Peter A.,Van Laeken Nancy,Carr Nick J.,Crawford Richard I.,Franzke Claus-Werner,Overall Christopher M.^ORCID,Granville David J.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-28070-0.pdf

Reference72 articles.

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2. Baum, S., Sakka, N., Artsi, O., Trau, H. & Barzilai, A. Diagnosis and classification of autoimmune blistering diseases. Autoimmun Rev 13, 482–489 (2014).

3. Jenne, D. E. & Tschopp, J. Granzymes, a family of serine proteases released from granules of cytolytic T lymphocytes upon T cell receptor stimulation. Immunol. Rev. 103, 53–71 (1988).

4. Isaaz, S., Baetz, K., Olsen, K., Podack, E. & Griffiths, G. M. Serial killing by cytotoxic T lymphocytes: T cell receptor triggers degranulation, re-filling of the lytic granules and secretion of lytic proteins via a non-granule pathway. Eur. J. Immunol. 25, 1071–1079 (1995).

5. Hiebert, P. R. & Granville, D. J. Granzyme B in injury, inflammation, and repair. Trends Mol Med 18, 732–741 (2012).

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