Fam64a is a novel cell cycle promoter of hypoxic fetal cardiomyocytes in mice
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-04823-1.pdf
Reference26 articles.
1. Puente, B. N. et al. The oxygen-rich postnatal environment induces cardiomyocyte cell-cycle arrest through DNA damage response. Cell. 157, 565–579 (2014).
2. Guimarães-Camboa, N. et al. HIF1α represses cell stress pathways to allow proliferation of hypoxic fetal cardiomyocytes. Dev Cell. 33, 507–521 (2015).
3. Leone, M., Magadum, A. & Engel, F. B. Cardiomyocyte proliferation in cardiac development and regeneration: a guide to methodologies and interpretations. Am J Physiol Heart Circ Physiol 309, H1237–H1250 (2015).
4. Senyo, S. E. et al. Mammalian heart renewal by pre-existing cardiomyocytes. Nature. 493, 433–436 (2013).
5. Hashimoto, H. et al. Time-lapse imaging of cell cycle dynamics during development in living cardiomyocyte. J Mol Cell Cardiol 72, 241–249 (2014).
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