Neurological sequel of chronic kidney disease: From diminished Acetylcholinesterase activity to mitochondrial dysfunctions, oxidative stress and inflammation in mice brain-Reference-Cited by-同舟云学术

Neurological sequel of chronic kidney disease: From diminished Acetylcholinesterase activity to mitochondrial dysfunctions, oxidative stress and inflammation in mice brain

Published:2019-02-28 Issue:1 Volume:9 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Mazumder Muhammed Khairujjaman,Paul Rajib,Bhattacharya Pallab,Borah Anupom

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-37935-3.pdf

Reference110 articles.

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2. Levey, A. S. et al. The definition, classification and prognosis of chronic kidney disease: a KDIGO Controversies Conference report. Kidney Int. 80, 17–28 (2011).

3. Chadban, S. J. et al. Prevalence of kidney damage in Australian adults: the AusDiab kidney study. J. Am. Soc. Nephrol. 14, S131–138 (2003).

4. Fox, C. S. et al. Cross-sectional association of kidney function with valvular and annular calcification: the Framingham heart study. J. Am. Soc. Nephrol. 17, 521–527 (2006).

5. Sakhuja, V. & Sud, K. End-stage renal disease in India and Pakistan: burden of disease and management issues. Kidney Int. 83, S115–118 (2003).

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