Histopathological Changes Caused by Inflammation and Oxidative Stress in Diet-Induced-Obese Mouse following Experimental Lung Injury-Reference-Cited by-同舟云学术

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Histopathological Changes Caused by Inflammation and Oxidative Stress in Diet-Induced-Obese Mouse following Experimental Lung Injury

Published:2018-09-24 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Wang Fengyuan,Zuo Zhicai,Chen Kejie,Fang Jing,Cui Hengmin,Shu Gang,Zhou Yi,Chen Zhengli,Huang Chao,Liu Wentao

Funder

Natural Science Foundation of Science and Technology Department of Sichuan Province

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-32420-3.pdf

Reference56 articles.

1. Toshima, M., Ohtani, Y. & Ohtani, O. Three-dimensional architecture of elastin and collagen fiber networks in the human and rat lung. Archives of Histology & Cytology 67, 31–40 (2004).

2. Marin, F., Luquet, G., Marie, B. & Medakovic, D. Molluscan shell proteins: primary structure, origin, and evolution. Current Topics in Developmental Biology 80, 209–276 (2008).

3. Lee, J. S. et al. TLR-4 pathway mediates the inflammatory response but not bacterial elimination in E. coli pneumonia. American Journal of Physiology Lung Cellular & Molecular Physiology 289, L731–L738 (2005).

4. Ong, E. S. et al. E. coli pneumonia induces CD18-independent airway neutrophil migration in the absence of increased lung vascular permeability. American Journal of Physiology Lung Cellular & Molecular Physiology 285, L879–L888 (2003).

5. Abraham, E. Neutrophils and acute lung injury. Critical Care Medicine 31, S195–199 (2003).

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