The gliadin peptide 31-43 exacerbates kainate neurotoxicity in epilepsy models
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-14845-4.pdf
Reference37 articles.
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2. Briani, C. et al. Neurological complications of celiac disease and autoimmune mechanisms: a prospective study. J. Neuroimmunol. 195(1-2), 171–5 (2008).
3. Hadjivassiliou, M. et al. Clinical, radiological, neurophysiological, and neuropathological characteristics of gluten ataxia. Lancet. 352(9140), 1582–5 (1998).
4. Bashiri, H., Afshari, D., Babaei, N. & Ghadami, M. R. Celiac Disease and Epilepsy: The Effect of Gluten-Free Diet on Seizure Control. Adv Clin. Exp. Med. 25(4), 751–4 (2016).
5. Mavroudi, A. et al. Increased prevalence of silent celiac disease among Greek epileptic children. Pediatr, Neurol. 36(3), 165–9 (2007).
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3. Interplay between Type 2 Transglutaminase (TG2), Gliadin Peptide 31-43 and Anti-TG2 Antibodies in Celiac Disease;International Journal of Molecular Sciences;2020-05-23
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5. Anticonvulsant and Neuroprotective Effects of the Thyroid Hormone Metabolite 3-Iodothyroacetic Acid;Thyroid;2018-10
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