Activation of the unfolded protein response in high glucose treated endothelial cells is mediated by methylglyoxal
Author:
Funder
University of Warwick | Warwick Medical School
Ministry of Education, Saudi Arabia
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-019-44358-1.pdf
Reference57 articles.
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2. Xue, M. et al. Activation of NF-E2-Related Factor-2 Reverses Biochemical Dysfunction of Endothelial Cells Induced by Hyperglycemia Linked to Vascular Disease. Diabetes 57, 2809–2817 (2008).
3. Nishikawa, T. et al. Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemia damage. Nature 404, 787–790 (2000).
4. Stratmann, B. et al. Glyoxalase 1-knockdown in human aortic endothelial cells – effect on the proteome and endothelial function estimates. Sci Rep 6, 37737 (2016).
5. Dobler, D., Ahmed, N., Song, L. J., Eboigbodin, K. E. & Thornalley, P. J. Increased dicarbonyl metabolism in endothelial cells in hyperglycemia induces anoikis and impairs angiogenesis by RGD and GFOGER motif modification. Diabetes 55, 1961–1969 (2006).
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