Angiotensin II-mediated MYH9 downregulation causes structural and functional podocyte injury in diabetic kidney disease-Reference-Cited by-同舟云学术

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Angiotensin II-mediated MYH9 downregulation causes structural and functional podocyte injury in diabetic kidney disease

Published:2019-05-22 Issue:1 Volume:9 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Kang Jeong Suk,Lee Seung Joo,Lee Ji-Hye,Kim Ji-Hee,Son Seung Seob,Cha Seung-Kuy,Lee Eun Soo,Chung Choon Hee,Lee Eun Young

Funder

National Research Foundation of Korea

Korea Health Industry Development Institute

Soonchunhyang University

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-019-44194-3.pdf

Reference56 articles.

1. Fayaz, A. H. & Axel, P. Role of oxidative stress in diabetic kidney disease. Med. Sci. Monit. 16, RA37–48 (2010).

2. Somlo, S. & Mundel, P. Getting a foothold in nephrotic syndrome. Nat. Genet. 24, 333–335 (2000).

3. Takeda, T., McQuistan, T., Orlando, R. A. & Farquhar, M. G. Loss of glomerular foot processes is associated with uncoupling of podocalyxin from the actin cytoskeleton. J. Clin. Invest. 108, 289–301 (2001).

4. Faul, C. et al. Actin up: regulation of podocyte structure and function by components of the actin cytoskeleton. Trends Cell Biol. 17, 428–437 (2007).

5. Winn, M. P. et al. A mutation in the TRPC6 cation channel causes familial focal segmental glomerulosclerosis. Science 308, 1801–1804 (2005).

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