Behavioral impairment in SHATI/NAT8L knockout mice via dysfunction of myelination development-Reference-Cited by-同舟云学术

Behavioral impairment in SHATI/NAT8L knockout mice via dysfunction of myelination development

Published:2017-12 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Sumi Kazuyuki,Uno Kyosuke,Noike Hiroshi,Tomohiro Takenori,Hatanaka Yasumaru,Furukawa-Hibi Yoko,Nabeshima Toshitaka,Miyamoto Yoshiaki,Nitta Atsumi^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-17151-1.pdf

Reference32 articles.

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2. Ariyannur, P. S. et al. Methamphetamine-induced neuronal protein NAT8L is the NAA biosynthetic enzyme: implications for specialized acetyl coenzyme A metabolism in the CNS. Brain. Res 1335, 1–13 (2010).

3. Wiame, E. et al. Molecular identification of aspartate N-acetyltransferase and its mutation in hypoacetylaspartia. Biochem J 425, 127–136 (2010).

4. Callicott, J. H. et al. Physiological dysfunction of the dorsolateral prefrontal cortex in schizophrenia revisited. Cereb Cortex 10, 1078–1092 (2000).

5. Howells, F. M. et al. 1H-magnetic resonance spectroscopy (1H-MRS) in methamphetamine dependence and methamphetamine induced psychosis. Schizophr Res 153, 122–128 (2014).

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