The PINK1 kinase-driven ubiquitin ligase Parkin promotes mitochondrial protein import through the presequence pathway in living cells
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-019-47352-9.pdf
Reference79 articles.
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2. Devi, L., Prabhu, B. M., Galati, D. F., Avadhani, N. G. & Anandatheerthavarada, H. K. Accumulation of amyloid precursor protein in the mitochondrial import channels of human Alzheimer’s disease brain is associated with mitochondrial dysfunction. J Neurosci 26, 9057–9068, https://doi.org/10.1523/JNEUROSCI.1469-06.2006 (2006).
3. Li, Q. et al. ALS-linked mutant superoxide dismutase 1 (SOD1) alters mitochondrial protein composition and decreases protein import. Proc Natl Acad Sci USA 107, 21146–21151, https://doi.org/10.1073/pnas.1014862107 (2010).
4. Bender, A. et al. TOM40 mediates mitochondrial dysfunction induced by alpha-synuclein accumulation in Parkinson’s disease. PLoS One 8, e62277, https://doi.org/10.1371/journal.pone.0062277 (2013).
5. Gottschalk, W. K. et al. The Broad Impact of TOM40 on Neurodegenerative Diseases in Aging. J Parkinsons Dis Alzheimers Dis 1, https://doi.org/10.13188/2376-922X.1000003 (2014).
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