Peripheral Expression of Mutant Huntingtin is a Critical Determinant of Weight Loss and Metabolic Disturbances in Huntington’s Disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-019-46470-8.pdf
Reference90 articles.
1. The Huntington’s Disease Collaborative Research Group. A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington’s disease chromosomes. Cell 72, 971–983 (1993).
2. Hoogeveen, A. T. et al. Characterization and localization of the Huntington disease gene product. Hum Mol Genet 2, 2069–2073 (1993).
3. Strong, T. V. et al. Widespread expression of the human and rat Huntington’s disease gene in brain and nonneural tissues. Nat Genet 5, 259–265, https://doi.org/10.1038/ng1193-259 (1993).
4. Marques Sousa, C. & Humbert, S. Huntingtin: here, there, everywhere! J Huntingtons Dis 2, 395–403, https://doi.org/10.3233/JHD-130082 (2013).
5. Sanberg, P. R., Fibiger, H. C. & Mark, R. F. Body weight and dietary factors in Huntington’s disease patients compared with matched controls. Med J Aust 1, 407–409 (1981).
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