RNA-Seq Analysis of Spinal Cord Tissues from hPFN1G118V Transgenic Mouse Model of ALS at Pre-symptomatic and End-Stages of Disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-31132-y.pdf
Reference39 articles.
1. Kiernan, M. C. et al. Amyotrophic lateral sclerosis. Lancet 377, 942–955, https://doi.org/10.1016/S0140-6736(10)61156-7 (2011).
2. Sreedharan, J. & Brown, R. H. Jr. Amyotrophic lateral sclerosis: Problems and prospects. Ann Neurol 74, 309–316, https://doi.org/10.1002/ana.24012 (2013).
3. Olsen, M. K. et al. Disease mechanisms revealed by transcription profiling in SOD1‐G93A transgenic mouse spinal cord. Annals of Neurology 50, 730–740, https://doi.org/10.1002/ana.1252 (2001).
4. de Oliveira, G. P., Alves, C. J. & Chadi, G. Early gene expression changes in spinal cord from SOD1(G93A) Amyotrophic Lateral Sclerosis animal model. Frontiers in Cellular Neuroscience 7, 216, https://doi.org/10.3389/fncel.2013.00216 (2013).
5. Perrin, F. E. et al. No widespread induction of cell death genes occurs in pure motoneurons in an amyotrophic lateral sclerosis mouse model. Human molecular genetics 14, 3309–3320, https://doi.org/10.1093/hmg/ddi357 (2005).
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