Interleukin-27 controls basal pain threshold in physiological and pathological conditions-Reference-Cited by-同舟云学术

Interleukin-27 controls basal pain threshold in physiological and pathological conditions

Published:2018-07-23 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Sasaguri Tomoko,Taguchi Toru,Murata Yuzo,Kobayashi Kimiko,Iizasa Sayaka,Iizasa Ei’ichi,Tsuda Makoto,Hirakawa Naomi,Hara Hiromitsu,Yoshida Hiroki,Yasaka Toshiharu

Funder

Japan Society for the Promotion of Science

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-29398-3.pdf

Reference67 articles.

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3. Gregory, N. S. et al. An overview of animal models of pain: disease models and outcome measures. J. Pain 14, 1255–1269, https://doi.org/10.1016/j.jpain.2013.06.008 (2013).

4. Kim, C. F. & Moalem-Taylor, G. Interleukin-17 contributes to neuroinflammation and neuropathic pain following peripheral nerve injury in mice. J. Pain 12, 370–383, https://doi.org/10.1016/j.jpain.2010.08.003 (2011).

5. Day, Y. J. et al. Lack of interleukin-17 leads to a modulated micro-environment and amelioration of mechanical hypersensitivity after peripheral nerve injury in mice. Pain 155, 1293–1302, https://doi.org/10.1016/j.pain.2014.04.004 (2014).

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