A link between plasma membrane calcium ATPase 2 (PMCA2), estrogen and estrogen receptor α signaling in mechanical pain-Reference-Cited by-同舟云学术

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A link between plasma membrane calcium ATPase 2 (PMCA2), estrogen and estrogen receptor α signaling in mechanical pain

Published:2018-11-22 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Khariv Veronika,Acioglu Cigdem,Ni Li,Ratnayake Ayomi,Li Lun,Tao Yuan-Xiang,Heary Robert F.,Elkabes Stella

Funder

Reynolds Family Spine Laboratory Funds

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-35263-0.pdf

Reference59 articles.

1. Mogil, J. S. & Belknap, J. K. Sex and Genotype Determine the Selective Activation of Neurochemically-Distinct Mechanisms of Swim Stress-Induced Analgesia. Pharmacol Biochem Behav 56, 61–66 (1997).

2. Joseph, E. K. & Levine, J. D. Sexual dimorphism in the contribution of protein kinase c isoforms to nociception in the streptozotocin diabetic rat. Neuroscience 120, 907–913 (2003).

3. Sorge, R. E. et al. Spinal cord Toll-like receptor 4 mediates inflammatory and neuropathic hypersensitivity in male but not female mice. J Neurosci 31, 15450–15454 (2011).

4. Khariv, V. et al. Impaired sensitivity to pain stimuli in plasma membrane calcium ATPase 2 (PMCA2) heterozygous mice: a possible modality- and sex-specific role for PMCA2 in nociception. FASEB J 31, 224–237 (2017).

5. Kurnellas, M. P., Nicot, A., Shull, G. E. & Elkabes, S. Plasma membrane calcium ATPase deficiency causes neuronal pathology in the spinal cord: a potential mechanism for neurodegeneration in multiple sclerosis and spinal cord injury. FASEB J 19, 298–300 (2005).

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