Anaerobiosis influences virulence properties of Pseudomonas aeruginosa cystic fibrosis isolates and the interaction with Staphylococcus aureus
Author:
Funder
The Humane Research Trust
RCUK | Biotechnology and Biological Sciences Research Council
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-019-42952-x.pdf
Reference100 articles.
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2. Welsh, M. J. & Smith, A. E. Molecular Mechanisms of Cftr Chloride Channel Dysfunction in Cystic-Fibrosis. Cell 73, 1251–1254, https://doi.org/10.1016/0092-8674(93)90353-R (1993).
3. Matsui, H. et al. Evidence for periciliary liquid layer depletion, not abnormal ion composition, in the pathogenesis of cystic fibrosis airways disease. Cell 95, 1005–1015 (1998).
4. Kreda, S. M., Davis, C. W. & Rose, M. C. CFTR, mucins, and mucus obstruction in cystic fibrosis. Cold Spring Harbor perspectives in medicine 2, a009589, https://doi.org/10.1101/cshperspect.a009589 (2012).
5. Williams, R. E. Healthy carriage of Staphylococcus aureus: its prevalence and importance. Bacteriol Rev 27, 56–71 (1963).
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