Triptolide attenuates proteinuria and podocyte apoptosis via inhibition of NF-κB/GADD45B
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-29203-1.pdf
Reference32 articles.
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2. Wiggins, R. C. The spectrum of podocytopathies: a unifying view of glomerular diseases. Kidney international 71, 1205–1214, https://doi.org/10.1038/sj.ki.5002222 (2007).
3. Shi, S. et al. Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis. Journal of the American Society of Nephrology: JASN 19, 2159–2169, https://doi.org/10.1681/asn.2008030312 (2008).
4. Chen, Z. et al. GADD45B mediates podocyte injury in zebrafish by activating the ROS-GADD45B-p38 pathway. Cell death & disease 7, e2068, https://doi.org/10.1038/cddis.2015.300 (2016).
5. Liu, Q. Triptolide and its expanding multiple pharmacological functions. International immunopharmacology 11, 377–383, https://doi.org/10.1016/j.intimp.2011.01.012 (2011).
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