CD38 promotes pristane-induced chronic inflammation and increases susceptibility to experimental lupus by an apoptosis-driven and TRPM2-dependent mechanism-Reference-Cited by-同舟云学术

CD38 promotes pristane-induced chronic inflammation and increases susceptibility to experimental lupus by an apoptosis-driven and TRPM2-dependent mechanism

Published:2018-02-20 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

García-Rodríguez Sonia,Rosal-Vela Antonio,Botta Davide^ORCID,Cumba Garcia Luz M.,Zumaquero Esther,Prados-Maniviesa Verónica,Cerezo-Wallis Daniela,Lo Buono Nicola^ORCID,Robles-Guirado José-Ángel,Guerrero Salvador,González-Paredes Elena,Andrés-León Eduardo^ORCID,Corbí Ángel,Mack Matthias,Koch-Nolte Friedrich,Merino Ramón,Zubiaur Mercedes,Lund Frances E.,Sancho Jaime^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-21337-6.pdf

Reference96 articles.

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3. Zhuang, H., Szeto, C., Han, S., Yang, L. & Reeves, W. H. Animal Models of Interferon Signature PositiveLupus. Frontiers in immunology 6, 291, https://doi.org/10.3389/fimmu.2015.00291 , scisignal.257mr1 (2015).

4. Quarona, V. et al. CD38 and CD157: A long journey from activation markers to multifunctional molecules. Cytometry B Clin Cytom 84, 207–217, https://doi.org/10.1002/cyto.b.21092 (2013).

5. Koch-Nolte, F., Haag, F., Guse, A. H., Lund, F. & Ziegler, M. Emerging roles of NAD+ and its metabolites in cell signaling. Sci Signal 2, mr1, https://doi.org/10.1126/scisignal.257mr1 (2009).

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