Prenatal hypoxia leads to hypertension, renal renin-angiotensin system activation and exacerbates salt-induced pathology in a sex-specific manner
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Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-08365-4.pdf
Reference50 articles.
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3. Hutter, D., Kingdom, J. & Jaeggi, E. Causes and mechanisms of intrauterine hypoxia and its impact on the fetal cardiovascular system: a review. Int J Pediatr 2010, 401323, doi: 10.1155/2010/401323 (2010).
4. Kingdom, J. C. & Kaufmann, P. Oxygen and placental villous development: origins of fetal hypoxia. Placenta 18, 613–621 (1997).
5. Peeters, L. L., Sheldon, R. E., Jones, M. D., Makowski, E. L. & Meschia, G. Blood flow to fetal organs as a function of arterial oxygen content. Am J Obstet Gynecol 135, 637–646 (1979).
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