Amplification mode differs along the length of the mouse cochlea as revealed by connexin 26 deletion from specific gap junctions
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-04279-3.pdf
Reference56 articles.
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3. Ashmore, J. Cochlear outer hair cell motility. Phys Rev 88, 173–210 (2008).
4. Mellado Lagarde, M. M., Drexl, M., Lukashkina, V. A., Lukashkin, A. N. & Russell, I. J. Outer hair cell somatic, not hair bundle, motility is the basis of the cochlear amplifier. Nature Neurosci 11, 746–748 (2008).
5. Weddell, T. D. et al. Prestin links extrinsic tuning to neural excitation in the mammalian cochlea. Curr Biol 21, R682–683 (2011).
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1. Preservation of developmental spontaneous activity enables early auditory system maturation in deaf mice;PLOS Biology;2023-06-27
2. A Gap-Junction Mutation Reveals That Outer Hair Cell Extracellular Receptor Potentials Drive High-Frequency Cochlear Amplification;The Journal of Neuroscience;2022-09-09
3. Connexin Mutations and Hereditary Diseases;International Journal of Molecular Sciences;2022-04-12
4. A gap-junction mutation in the mouse cochlea reveals cochlear amplification is driven by outer hair cell extracellular receptor potentials;2021-08-12
5. Structure and Function of Cochlear Gap Junctions and Implications for the Translation of Cochlear Gene Therapies;Frontiers in Cellular Neuroscience;2019-11-27
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