Metformin prevention of doxorubicin resistance in MCF-7 and MDA-MB-231 involves oxidative stress generation and modulation of cell adaptation genes-Reference-Cited by-同舟云学术

Metformin prevention of doxorubicin resistance in MCF-7 and MDA-MB-231 involves oxidative stress generation and modulation of cell adaptation genes

Published:2019-04-10 Issue:1 Volume:9 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Marinello Poliana Camila,Panis Carolina,Silva Thamara Nishida Xavier,Binato Renata,Abdelhay Eliana,Rodrigues Juliana Alves,Mencalha André Luiz,Lopes Natália Medeiros Dias,Luiz Rodrigo Cabral,Cecchini Rubens,Cecchini Alessandra Lourenço

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-019-42357-w.pdf

Reference56 articles.

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2. Panis, C. et al. Oxidative stress and hematological profiles of advanced breast cancer patients subjected to paclitaxel or doxorubicin chemotherapy. Breast Cancer Res Treat. 133(1), 89–97 (2012).

3. Zhang, M., Wang, J., Zhang, L. & Zhou, S. EGFR-Nrf2 pathway plays a role in cancer cell’s chemoresistance. Bioscience Hypotheses 2, 261–263 (2009).

4. Dalmases, A. et al. Deficiency in p53 is required for doxorubicin induced transcriptional activation of NF-кB target genes in human breast cancer. Oncotarget, 5 (2013).

5. Ingles-Esteve, J. et al. Inhibition of specific NF-kappaB activity contributes to the tumor suppressor function of 14-3-3sigma in breast cancer. PLoS One 7, e38347 (2012).

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