Cbl-mediated K63-linked ubiquitination of JAK2 enhances JAK2 phosphorylation and signal transduction
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-04078-w.pdf
Reference48 articles.
1. Imada, K. & Leonard, W. J. The Jak-STAT pathway. Mol Immunol 37, 1–11 (2000).
2. Gadina, M. et al. Signaling by type I and II cytokine receptors: ten years after. Curr Opin Immunol 13, 363–373 (2001).
3. Tefferi, A. et al. Proposals and rationale for revision of the World Health Organization diagnostic criteria for polycythemia vera, essential thrombocythemia, and primary myelofibrosis: recommendations from an ad hoc international expert panel. Blood 110, 1092–1097, doi: 10.1182/blood-2007-04-083501 (2007).
4. Kralovics, R. et al. A gain-of-function mutation of JAK2 in myeloproliferative disorders. N Engl J Med 352, 1779–1790, doi: 10.1056/NEJMoa051113 (2005).
5. Baxter, E. J. et al. Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders. Lancet 365, 1054–1061, doi: 10.1016/S0140-6736(05)71142-9 (2005).
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