Embryonic defence mechanisms against glucose-dependent oxidative stress require enhanced expression of Alx3 to prevent malformations during diabetic pregnancy-Reference-Cited by-同舟云学术

Embryonic defence mechanisms against glucose-dependent oxidative stress require enhanced expression of Alx3 to prevent malformations during diabetic pregnancy

Published:2017-03-24 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

García-Sanz Patricia,Mirasierra Mercedes,Moratalla Rosario,Vallejo Mario

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-00334-1.pdf

Reference70 articles.

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3. Martínez-Frías, M. L. Epidemiological analysis of outcomes of pregnancy in diabetic mothers: identification of the most characteristic and most frequent congenital anomalies. Am. J. Med. Genet. 51, 108–113 (1994).

4. Simeone, R. M. et al. Diabetes and congenital heart defects: a systematic review, meta-analysis, and modeling project. Am J Prev Med 48, 195–204 (2015).

5. Fine, E. L., Horal, M., Chang, T. I., Fortin, G. & Loeken, M. R. Evidence that elevated glucose causes altered gene expression, apoptosis, and neural tube defects in a mouse model of diabetic pregnancy. Diabetes 48, 2454–2462 (1999).

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