Interplay of DNA methyltransferase 1 and EZH2 through inactivation of Stat3 contributes to β-elemene-inhibited growth of nasopharyngeal carcinoma cells
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-00626-6.pdf
Reference72 articles.
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2. Hildesheim, A. & Wang, C. P. Genetic predisposition factors and nasopharyngeal carcinoma risk: a review of epidemiological association studies, 2000–2011: Rosetta Stone for NPC: genetics, viral infection, and other environmental factors. Semin Cancer Biol 22, 107–116 (2012).
3. Yang, G. et al. The inhibitory effects of extracellular ATP on the growth of nasopharyngeal carcinoma cells via P2Y2 receptor and osteopontin. J Exp Clin Cancer Res 33, 53 (2014).
4. Bruce, J. P., Yip, K., Bratman, S. V., Ito, E. & Liu, F. F. Nasopharyngeal Cancer: Molecular Landscape. J Clin Oncol 33, 3346–3355 (2015).
5. Wang, S., Zhang, R., Claret, F. X. & Yang, H. Involvement of microRNA-24 and DNA methylation in resistance of nasopharyngeal carcinoma to ionizing radiation. Mol Cancer Ther 13, 3163–3174 (2014).
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