Endothelial Dysfunction in Severe Preeclampsia is Mediated by Soluble Factors, Rather than Extracellular Vesicles
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-06178-z.pdf
Reference45 articles.
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3. Walker, M. G. et al. Sex-specific basis of severe placental dysfunction leading to extreme preterm delivery. Placenta 33, 568–571, doi: 10.1016/j.placenta.2012.03.011 (2012).
4. Redline, R. W. Classification of placental lesions. American journal of obstetrics and gynecology 213, S21–28, doi: 10.1016/j.ajog.2015.05.056 (2015).
5. Rajakumar, A. et al. Transcriptionally active syncytial aggregates in the maternal circulation may contribute to circulating soluble fms-like tyrosine kinase 1 in preeclampsia. Hypertension 59, 256–264, doi: 10.1161/HYPERTENSIONAHA.111.182170 (2012).
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