Whole body and hematopoietic ADAM8 deficiency does not influence advanced atherosclerotic lesion development, despite its association with human plaque progression-Reference-Cited by-同舟云学术

Whole body and hematopoietic ADAM8 deficiency does not influence advanced atherosclerotic lesion development, despite its association with human plaque progression

Published:2017-09-15 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Theodorou Kosta^ORCID,van der Vorst Emiel P. C.,Gijbels Marion J.,Wolfs Ine M. J.,Jeurissen Mike,Theelen Thomas L.,Sluimer Judith C.,Wijnands Erwin,Cleutjens Jack P.,Li Yu,Jansen Yvonne,Weber Christian,Ludwig Andreas,Bentzon Jacob F.,Bartsch Jörg W.^ORCID,Biessen Erik A. L.,Donners Marjo M. P. C.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-10549-x.pdf

Reference50 articles.

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3. Dreymueller, D., Pruessmeyer, J., Groth, E. & Ludwig, A. The role of ADAM-mediated shedding in vascular biology. European journal of cell biology 91, 472–485, doi: 10.1016/j.ejcb.2011.09.003 (2012).

4. van der Vorst, E. P. et al. Myeloid A disintegrin and metalloproteinase domain 10 deficiency modulates atherosclerotic plaque composition by shifting the balance from inflammation toward fibrosis. The American journal of pathology 185, 1145–1155, doi: 10.1016/j.ajpath.2014.11.028 (2015).

5. Sun, C., Wu, M. H., Lee, E. S. & Yuan, S. Y. A disintegrin and metalloproteinase 15 contributes to atherosclerosis by mediating endothelial barrier dysfunction via Src family kinase activity. Arterioscler Thromb Vasc Biol 32, 2444–2451, doi: 10.1161/ATVBAHA.112.252205 (2012).

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