Copper(I)-binding properties of de-coppering drugs for the treatment of Wilson disease. α-Lipoic acid as a potential anti-copper agent
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Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-19873-2.pdf
Reference30 articles.
1. Bull, P. C., Thomas, G. R., Rommens, J. M., Forbes, J. R. & Cox, D. W. The Wilson disease gene is a putative copper transporting P-type ATPase similar to the Menkes gene. Nat Genet 5, 327–337, https://doi.org/10.1038/ng1293-327 (1993).
2. Tanzi, R. E. et al. The Wilson disease gene is a copper transporting ATPase with homology to the Menkes disease gene. Nat Genet 5, 344–350, https://doi.org/10.1038/ng1293-344 (1993).
3. Terada, K. et al. Copper incorporation into ceruloplasmin in rat livers. Biochim Biophys Acta 1270, 58–62 (1995).
4. Terada, K. et al. Restoration of holoceruloplasmin synthesis in LEC rat after infusion of recombinant adenovirus bearing WND cDNA. J Biol Chem 273, 1815–1820 (1998).
5. Terada, K. et al. Biliary excretion of copper in LEC rat after introduction of copper transporting P-type ATPase, ATP7B. FEBS Lett 448, 53–56 (1999).
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