Roles of aberrant hemichannel activities due to mutant connexin26 in the pathogenesis of KID syndrome-Reference-Cited by-同舟云学术

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Roles of aberrant hemichannel activities due to mutant connexin26 in the pathogenesis of KID syndrome

Published:2018-08-27 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Taki T.^ORCID,Takeichi T.,Sugiura K.,Akiyama M.

Funder

Japan Society for the Promotion of Science

Lydia O’leary Memorial Pias Dermatological Foundation Kanae Foundation for the Promotion of Medical Science

Japan Agency for Medical Research and Development

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-30757-3.pdf

Reference59 articles.

1. Skinner, B. A., Greist, M. C. & Norins, A. L. The keratitis, ichthyosis, and deafness (KID) syndrome. Arch Dermatol 117, 285–289 (1981).

2. Caceres-Rios, H., Tamayo-Sanchez, L., Duran-Mckinster, C., de la Luz Orozco, M. & Ruiz-Maldonado, R. Keratitis, ichthyosis, and deafness (KID syndrome): review of the literature and proposal of a new terminology. Pediatr Dermatol 13, 105–113 (1996).

3. Gilliam, A. & Williams, M. L. Fatal septicemia in an infant with keratitis, ichthyosis, and deafness (KID) syndrome. Pediatr Dermatol 19, 232–236 (2002).

4. Miteva, L. Keratitis, ichthyosis, and deafness (KID) syndrome. Pediatr Dermatol 19, 513–516 (2002).

5. Zhang, X., He, Y., Zhou, H., Luo, Q. & Li, C. Severe ichthyosis-related disorders in children: response to acitretin. J Dermatolog Treat 18, 118–122, https://doi.org/10.1080/09546630601156348 (2007).

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