USP9X deubiquitylating enzyme maintains RAPTOR protein levels, mTORC1 signalling and proliferation in neural progenitors-Reference-Cited by-同舟云学术

USP9X deubiquitylating enzyme maintains RAPTOR protein levels, mTORC1 signalling and proliferation in neural progenitors

Published:2017-03-24 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Bridges Caitlin R.,Tan Men-Chee,Premarathne Susitha,Nanayakkara Devathri,Bellette Bernadette,Zencak Dusan,Domingo Deepti,Gecz Jozef^ORCID,Murtaza Mariyam,Jolly Lachlan A.,Wood Stephen A.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-00149-0.pdf

Reference46 articles.

1. Blomen, V. A. & Boonstra, J. Cell fate determination during G1 phase progression. Cellular and Molecular Life Sciences 64, 3084–3104, doi: 10.1007/s00018-007-7271-z (2007).

2. Fingar, D. C. et al. mTOR Controls Cell Cycle Progression through Its Cell Growth Effectors S6K1 and 4E-BP1/Eukaryotic Translation Initiation Factor 4E. Molecular and Cellular Biology 24, 200–216, doi: 10.1128/mcb.24.1.200-216.2004 (2004).

3. Dibble, C. C. & Manning, B. D. Signal integration by mTORC1 coordinates nutrient input with biosynthetic output. Nat Cell Biol 15, 555–564, doi: 10.1038/ncb2763 (2013).

4. Magri, L. & Galli, R. mTOR signaling in neural stem cells: from basic biology to disease. Cell. Mol. Life Sci., 1-12, 10.1007/s00018-012-1196-x (2012).

5. Scheffer, I. E. et al. Mutations in mammalian target of rapamycin regulator DEPDC5 cause focal epilepsy with brain malformations. Annals of Neurology 75, 782–787, doi: 10.1002/ana.24126 (2014).

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