Assessment of Pharmacology, Safety, and Metabolic activity of Capsaicin Feeding in Mice
Author:
Funder
U.S. Department of Health & Human Services | National Institutes of Health
American Heart Association
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-019-45050-0.pdf
Reference65 articles.
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2. Allegri, M. et al. A pharmacological treatment algorithm for localized neuropathic pain. Curr Med Res Opin 32, 377–384, https://doi.org/10.1185/03007995.2015.1129321 (2016).
3. Hoper, J., Helfert, S., Heskamp, M. L., Maihofner, C. G. & Baron, R. High concentration capsaicin for treatment of peripheral neuropathic pain: effect on somatosensory symptoms and identification of treatment responders. Curr Med Res Opin 30, 565–574, https://doi.org/10.1185/03007995.2013.869491 (2014).
4. Takahashi, N. et al. Neuronal TRPV1 activation regulates alveolar bone resorption by suppressing osteoclastogenesis via CGRP. Sci Rep 6, 29294, https://doi.org/10.1038/srep29294 (2016).
5. Chen, K. S. et al. Capsaicin protects endothelial cells and macrophage against oxidized low-density lipoprotein-induced injury by direct antioxidant action. Chem Biol Interact 228, 35–45, https://doi.org/10.1016/j.cbi.2015.01.007 (2015).
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