Targeting Mitochondrial Oxidative Phosphorylation Abrogated Irinotecan Resistance in NSCLC-Reference-Cited by-同舟云学术

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Targeting Mitochondrial Oxidative Phosphorylation Abrogated Irinotecan Resistance in NSCLC

Published:2018-10-24 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Lee Soohyun,Lee Jae-Seon,Seo Jinho,Lee Seon-Hyeong,Kang Joon Hee,Song Jaewhan,Kim Soo-Youl

Funder

National Research Foundation of Korea

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-33667-6.pdf

Reference23 articles.

1. Dempke, W. C. Targeted Therapy for NSCLC–A Double-edged Sword? Anticancer Res 35, 2503–2512 (2015).

2. Morrissy, A. S. et al. Divergent clonal selection dominates medulloblastoma at recurrence. Nature 529, 351–357, https://doi.org/10.1038/nature16478 (2016).

3. McGranahan, N. & Swanton, C. Clonal Heterogeneity and Tumor Evolution: Past, Present, and the Future. Cell 168, 613–628, https://doi.org/10.1016/j.cell.2017.01.018 (2017).

4. Farge, T. et al. Chemotherapy-Resistant Human Acute Myeloid Leukemia Cells Are Not Enriched for Leukemic Stem Cells but Require Oxidative Metabolism. Cancer Discov 7, 716–735, https://doi.org/10.1158/2159-8290.CD-16-0441 (2017).

5. Muus, P. et al. 1-beta-D-arabinofuranosylcytosine (Ara-C) enhances mitochondrial activities in human leukaemic cells. Br J Cancer 64, 29–34 (1991).

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