Hemopoietic Cell Kinase amplification with Protein Tyrosine Phosphatase Receptor T depletion leads to polycythemia, aberrant marrow erythoid maturation, and splenomegaly
Author:
Funder
Cancer Council Victoria
Leukaemia Foundation
Department of Health | National Health and Medical Research Council
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-019-43373-6.pdf
Reference35 articles.
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2. Bench, A. J. et al. Chromosome 20 deletions in myeloid malignancies: reduction of the common deleted region, generation of a PAC/BAC contig and identification of candidate genes. UK Cancer Cytogenetics Group (UKCCG). Oncogene 19(34), 3902–3913 (2000).
3. Fenaux, P., Morel, P. & Lai, J. L. Cytogenetics of myelodysplastic syndromes. Semin Hematol. 33(2), 127–138 (1996).
4. Heim, S. & Mitelman, F. Cytogenetic analysis in the diagnosis of acute leukemia. Cancer 70(6 suppl), 1701–1709 (1992).
5. MacKinnon, R. N. et al. The paradox of 20q11.21 amplification in a subset of cases of myeloid malignancy with chromosome 20 deletion. Genes, Chromosomes and Cancer 49, 998–1013, https://doi.org/10.1002/gcc.20806 (2010).
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