Estrogen attenuates AGTR1 expression to reduce pancreatic β-cell death from high glucose
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-15237-4.pdf
Reference45 articles.
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2. Butler, A. et al. Beta-cell deficit and increased beta-cell apoptosis in humans with type 2 diabetes. Diabetes. 52, 102–110 (2003).
3. Robertson, R. P. Chronic oxidative stress as a central mechanism for glucose toxicity in pancreatic islet beta cells in diabetes. J Biol Chem 279, 42351–42354 (2004).
4. Evans, J. L., Goldfine, I. D., Maddux, B. A. & Grodsky, G. M. Oxidative stress and stress-activated signaling pathways: a unifying hypothesis of type 2 diabetes. Endocr Rev 23, 599–622 (2002).
5. Andreyev, A., Kushnareva, Y. & Starkov, A. Mitochondrial metabolism of reactive oxygen species. Biochemistry (Mosc). 70, 200–214 (2005).
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