PMCA-replicated PrPD in urine of vCJD patients maintains infectivity and strain characteristics of brain PrPD: Transmission study-Reference-Cited by-同舟云学术

PMCA-replicated PrPD in urine of vCJD patients maintains infectivity and strain characteristics of brain PrPD: Transmission study

Published:2019-03-26 Issue:1 Volume:9 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Cali Ignazio,Lavrich Jody,Moda Fabio,Kofskey Diane,Nemani Satish Kumar,Appleby Brian,Tagliavini Fabrizio,Soto Claudio,Gambetti Pierluigi,Notari Silvio

Funder

U.S. Department of Health & Human Services | NIH | National Institute of Allergy and Infectious Diseases

U.S. Department of Health & Human Services | NIH | National Institute of Neurological Disorders and Stroke

Britton Fund

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-019-41694-0.pdf

Reference32 articles.

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2. Ritchie, D. L. & Ironside, J. W. Neuropathology of Human Prion Diseases. Prog Mol Biol Transl Sci 150, 319–339 (2017).

3. Head, M. W. et al. Peripheral tissue involvement in sporadic, iatrogenic, and variant Creutzfeldt-Jakob disease: an immunohistochemical, quantitative, and biochemical study. Am J Pathol 164, 143–153 (2004).

4. Bruce, M. E. et al. Transmissions to mice indicate that ‘new variant’ CJD is caused by the BSE agent. Nature 389, 498–501 (1997).

5. Hill, A. F. et al. The same prion strain causes vCJD and BSE. Nature 389, 448–450 (1997).

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