The interleukin-33-mediated inhibition of expression of two key genes implicated in atherosclerosis in human macrophages requires MAP kinase, phosphoinositide 3-kinase and nuclear factor-κB signaling pathways-Reference-Cited by-同舟云学术

The interleukin-33-mediated inhibition of expression of two key genes implicated in atherosclerosis in human macrophages requires MAP kinase, phosphoinositide 3-kinase and nuclear factor-κB signaling pathways

Published:2019-08-05 Issue:1 Volume:9 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Buckley Melanie L.,Williams Jessica O.,Chan Yee-Hung,Laubertová Lucia,Gallagher Hayley,Moss Joe W. E.,Ramji Dipak P.

Funder

RCUK | Medical Research Council

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-019-47620-8.pdf

Reference58 articles.

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2. Buckley, M. L. & Ramji, D. P. The influence of dysfunctional signaling and lipid homeostasis in mediating the inflammatory responses during atherosclerosis. Biochim Biophys Acta 1852, 1498–1510, https://doi.org/10.1016/j.bbadis.2015.04.011 (2015).

3. Moore, K. J., Sheedy, F. J. & Fisher, E. A. Macrophages in atherosclerosis: a dynamic balance. Nat Rev Immunol 13, 709–721, https://doi.org/10.1038/nri3520 (2013).

4. Michael, D. R., Ashlin, T. G., Buckley, M. L. & Ramji, D. P. Macrophages, lipid metabolism and gene expression in atherogenesis: a therapeutic target of the future? Clin Lipidol 7, 37–48, https://doi.org/10.2217/CLP.11.73 (2012).

5. Michael, D. R. et al. Differential regulation of macropinocytosis in macrophages by cytokines: Implications for foam cell formation and atherosclerosis. Cytokine 64, 357–361, https://doi.org/10.1016/j.cyto.2013.05.016 (2013).

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