The Role of Angiotensin II in Glomerular Volume Dynamics and Podocyte Calcium Handling
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-00406-2.pdf
Reference60 articles.
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2. Winn, M. P. et al. A mutation in the TRPC6 cation channel causes familial focal segmental glomerulosclerosis. Science 308, 1801–1804 (2005).
3. Winn, M. P., Daskalakis, N., Spurney, R. F. & Middleton, J. P. Unexpected role of TRPC6 channel in familial nephrotic syndrome: does it have clinical implications? J. Am. Soc. Nephrol. 17, 378–387 (2006).
4. Krall, P. et al. Podocyte-specific overexpression of wild type or mutant trpc6 in mice is sufficient to cause glomerular disease. PLoS ONE 5, e12859 (2010).
5. Wang, L. et al. Gq signaling causes glomerular injury by activating TRPC6. J. Clin. Invest. 125, 1913-26 (2015).
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