The immune-inflammation factor is associated with diabetic nephropathy: evidence from NHANES 2013–2018 and GEO database
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Publisher
Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s41598-024-68347-1.pdf
Reference33 articles.
1. Li, X. et al. Epigenetics in the pathogenesis of diabetic nephropathy. Acta Biochim. Biophys. Sin. (Shanghai) 54, 163–172. https://doi.org/10.3724/abbs.2021016 (2022).
2. Chen, J., Liu, Q., He, J. & Li, Y. Immune responses in diabetic nephropathy: Pathogenic mechanisms and therapeutic target. Front. Immunol. 13, 958790. https://doi.org/10.3389/fimmu.2022.958790 (2022).
3. Geng, K. et al. High glucose-induced STING activation inhibits diabetic wound healing through promoting M1 polarization of macrophages. Cell Death Discov. 9, 136. https://doi.org/10.1038/s41420-023-01425-x (2023).
4. Geng, K. et al. Innate immunity in diabetic wound healing: focus on the mastermind hidden in chronic inflammatory. Front. Pharmacol. 12, 653940. https://doi.org/10.3389/fphar.2021.653940 (2021).
5. Zhou, H., Ni, W. J., Meng, X. M. & Tang, L. Q. MicroRNAs as regulators of immune and inflammatory responses: Potential therapeutic targets in diabetic nephropathy. Front. Cell Dev. Biol. 8, 618536. https://doi.org/10.3389/fcell.2020.618536 (2020).
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