Knock-down of AHCY and depletion of adenosine induces DNA damage and cell cycle arrest
Author:
Funder
National Research Foundation of Korea
EC | Seventh Framework Programme
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-32356-8.pdf
Reference58 articles.
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2. Borchardt, R. T., Huber, J. A. & Wu, Y. S. Potential inhibitors of S-adenosylmethionine-dependent methyltransferases. 4. Further modifications of the amino acid and base portions of S-adenosyl-L-homocysteine. Journal of Medicinal Chemistry 19(9), 1094–1099 (1976).
3. Loehrer, F. M., Angst, C. P., Brunner, F. P., Haefeli, W. E. & Fowler, B. Evidence for disturbed S-adenosylmethionine: S-adenosylhomocysteine ratio in patients with end-stage renal failure: a cause for disturbed methylation reactions? Nephrol Dial Transplant. 13, 656–661 (1998).
4. Caudill, M. A. et al. Intracellular S-adenosylhomocysteine concentrations predict global DNA hypomethylation in tissues of methyl-deficient cystathionine beta-synthase heterozygous mice. The Journal of Nutrition 131(11), 2811–8 (2001).
5. Barić, I. et al. S-adenosylhomocysteine hydrolase deficiency in a human: A genetic disorder of methionine metabolism. Proceedings of the National Academy of Sciences 101(12), 4234–4239 (2004).
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