Distinct Phenotypes Induced by Three Degrees of Transverse Aortic Constriction in Mice
Author:
Funder
U.S. Department of Health & Human Services | National Institutes of Health
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-019-42209-7.pdf
Reference38 articles.
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3. Shah, S. J. et al. Phenotype-Specific Treatment of Heart Failure With Preserved Ejection Fraction: A Multiorgan Roadmap. Circulation 134, 73–90, https://doi.org/10.1161/CIRCULATIONAHA.116.021884 (2016).
4. Kalogeropoulos, A. P. et al. Characteristics and Outcomes of Adult Outpatients With Heart Failure and Improved or Recovered Ejection Fraction. JAMA Cardiol 1, 510–518, https://doi.org/10.1001/jamacardio.2016.1325 (2016).
5. Yu, Y., Hu, Z., Li, B., Wang, Z. & Chen, S. Ivabradine improved left ventricular function and pressure overload-induced cardiomyocyte apoptosis in a transverse aortic constriction mouse model. Mol Cell Biochem, https://doi.org/10.1007/s11010-018-3369-x (2018).
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